Search results for "Sickle cell trait"

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α-Thalassemia Does Not Seem to Influence Erythrocyte Deformability in Sickle Cell Trait Carriers

2014

Studies dealing with rheological red blood cell (RBC) behavior in sickle cell trait carriers are scarce. Moreover, the association with α-thalassemia (α-thal), which also modifies erythrocyte behavior, has not always been taken into account. We analyzed erythrocyte deformability by means of a shear stress diffractometer, along with hematological and biochemical parameters (glucose and plasma lipids), given their possible influence on erythrocyte deformability, in 14 sickle cell trait carriers and 23 healthy controls. Nine patients were also α-thal carriers and five were not. Among the thalassemia carriers, eight were heterozygous and one was homozygous. When compared with controls, sickle c…

AdultErythrocyte IndicesMalemedicine.medical_specialtyErythrocytesThalassemiaHemoglobin SickleClinical BiochemistryMutation MissenseSickle Cell Traitalpha-ThalassemiaErythrocyte DeformabilityInternal medicinePlasma lipidsmedicineHumansErythrocyte deformabilityMean corpuscular volumeGenetics (clinical)Sickle cell traitmedicine.diagnostic_testbusiness.industryBiochemistry (medical)HematologyMiddle Agedmedicine.diseaseRed blood cellHemoglobinopathymedicine.anatomical_structureEndocrinologyAmino Acid SubstitutionImmunologyFemaleHemoglobinbusinessHemoglobin
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Thalassemia (Microkarterocytosis) and Drepanocytosis their Forms and Genetics

1952

SummaryThalassemia, that I propose to call Microkarterocytosis, can be divided into three forms to be distinguished under the names of minima (healthy carriers), minor (Rietti and Greppi's disease), major (Cooley disease).It can be inferred that Thalassemia is a mutation developped in a mediterranean superior paleolitic race that showed also the character high cheek bones.In my researches of 1941-42 I for first showed that thalassemia is a dominant hereditary character with homozigotic letal effect (heterozigotes the healthy carriers, homozigotes Cooley' patients).Heterozigotes throug an increased expression of the morbid gene develop Rietti and Greppi disease, as I inferred in 1949.The res…

GeneticsHereditybusiness.industryAnemiaThalassemiabeta-ThalassemiaAnemia Sickle CellGeneral MedicineDiseasemedicine.diseaseSickle cell anemiaSickle Cell TraitRace (biology)Cheek bonesHumansThalassemiaMedicineAnemia sickle-cellbusinessActa geneticae medicae et gemellologiae
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